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Registro completo
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Biblioteca (s) : |
INIA Las Brujas. |
Fecha : |
23/02/2021 |
Actualizado : |
23/02/2021 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Autor : |
ARTIGAS, R.; FEDERICI, M.; VÁZQUEZ, N.; ALCÁNTARA, M.; RAMÍREZ, M.; GUERRA, S.; DUTRA, F.; LLAMBÍ, S. |
Afiliación : |
RODY ARTIGAS SILVA, Departamento de Genética y Mejora Animal, Área Genética. Facultad de Veterinaria, Universidad de la República (UdelaR). Montevideo, Uruguay; MARIA TERESA FEDERICI RODRIGUEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; NOELIA VÁZQUEZ ODO, Departamento de Morfología y Desarrollo, Área Anatomía. Facultad de Veterinaria, UdelaR. Montevideo, Uruguay; MARIANA ALCÁNTARA CRAPUCHET, Departamento de Genética y Mejora Animal, Área Genética. Facultad de Veterinaria, Universidad de la República (UdelaR). Montevideo, Uruguay.; MARIANA RAMIREZ FIRPO, Departamento de Genética y Mejora Animal, Área Genética. Facultad de Veterinaria, Universidad de la República (UdelaR). Montevideo, Uruguay.; SOFÍA GUERRA MARTÍNEZ, Departamento de Genética y Mejora Animal, Área Genética. Facultad de Veterinaria, Universidad de la República (UdelaR). Montevideo, Uruguay.; FERNANDO DUTRA QUINTANELA, DILAVE Miguel C. Rubino, Laboratorio Regional Este, Treinta y Tres, Uruguay.; SILVIA LLAMBÍ DELLACASA, Departamento de Genética y Mejora Animal, Área Genética. Facultad de Veterinaria, Universidad de la República (UdelaR). Montevideo, Uruguay. |
Título : |
Identificación por catálogo y detección molecular de bovinos Holstein portadores de braquiespina en Uruguay. [Identification through sire catalogues analysis and molecular detection of brachyspina carriers in Uruguayan Holstein.] |
Complemento del título : |
Artículo original. |
Fecha de publicación : |
2020 |
Fuente / Imprenta : |
FAVE Sección Ciencias Veterinarias, 2020, volume 19 (2), 50-54. OPEN ACCESS. DOI: https://doi.org/10.14409/favecv.v19i2.9523 |
ISSN : |
1666-938X (impresa); 2362-5589 (digital) |
DOI : |
10.14409/favecv.v19i2.9523 |
Idioma : |
Español |
Notas : |
Article history: Recibido 3 Marzo 2020; Aceptado 1 Agosto 2020; Disponible en línea 14 Agosto 2020.
Editor: P. Beldomenico.
Esta obra está bajo licencia internacional Creative Commons Reconocimiento-NoComercial-CompartirIgual 4.0. |
Contenido : |
RESUMEN. El síndrome de braquiespina es una condición genética de la raza Holstein, detectada en el año 2006. Es causado por una deleción de 3.3 Kb en el gen FANCI localizado en el cromosoma bovino 21. La mutación fue identificada en poblaciones de Holstein de Europa, América del Norte y Asia. Dada la importancia económica del defecto y su amplia distribución mundial, el objetivo de este trabajo ha sido la identificación de animales portadores en el núcleo de selección genética de la raza en Uruguay y el diagnóstico molecular del alelo deletéreo en animales del rodeo nacional. En el presente estudio se analizaron 2598 registros de toros Holstein del catálogo de padres del sistema de evaluación genética lechera, los registros de toros pertenecientes a los catálogos de semen Holstein disponible para Uruguay de los años 2014 al 2018; y 71 vacas pertenecientes al rodeo general. Se encontraron 28 toros portadores de braquiespina de un total de 377 toros con información genética del catálogo de padres y cuatro vacas portadoras de un total de 71 genotipificadas en nuestro laboratorio. Se demostró una disminución en el ingreso de semen de animales portadores al país entre los años 2014 y 2018. La frecuencia significativa de animales portadores en Uruguay evidencia la necesidad de implementar estrategias que permitan eliminar gradualmente el defecto de la población.
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SUMMARY. Brachyspina syndrome is a hereditary recessive disease of recent identification in the Holstein breed. It is caused by a deletion of 3.3Kb in the FANCI gene located in the bovine chromosome 21. The mutation was identified in Holstein populations of Europe, North America and Asia. Given the economic importance of the defect and its wide distribution, the objective of this work was the identification of carrier animals in the genetic selection nucleus of the breed in Uruguay and the molecular verification of the deleterious allele in animals of the national herd. In the present study, 2598 records of Holstein bulls were analyzed from the list of parents of the dairy genetic evaluation system, records of bulls belonging to the Holstein semen catalogs available for Uruguay from 2014 to 2018; and 71 cows belonging to the general herd. Twenty-eight brachyspina carrier bulls were found of a total of 377 bulls with genetic information from the list of parents and four carrier cows of a total of 71 genotyped in our laboratory. A decrease in the income of semen from carrier animals to the country between 2014 and 2018 was demonstrated. The significant frequency of carrier animals in Uruguay evidences the need to implement strategies to gradually eliminate the population defect. MenosRESUMEN. El síndrome de braquiespina es una condición genética de la raza Holstein, detectada en el año 2006. Es causado por una deleción de 3.3 Kb en el gen FANCI localizado en el cromosoma bovino 21. La mutación fue identificada en poblaciones de Holstein de Europa, América del Norte y Asia. Dada la importancia económica del defecto y su amplia distribución mundial, el objetivo de este trabajo ha sido la identificación de animales portadores en el núcleo de selección genética de la raza en Uruguay y el diagnóstico molecular del alelo deletéreo en animales del rodeo nacional. En el presente estudio se analizaron 2598 registros de toros Holstein del catálogo de padres del sistema de evaluación genética lechera, los registros de toros pertenecientes a los catálogos de semen Holstein disponible para Uruguay de los años 2014 al 2018; y 71 vacas pertenecientes al rodeo general. Se encontraron 28 toros portadores de braquiespina de un total de 377 toros con información genética del catálogo de padres y cuatro vacas portadoras de un total de 71 genotipificadas en nuestro laboratorio. Se demostró una disminución en el ingreso de semen de animales portadores al país entre los años 2014 y 2018. La frecuencia significativa de animales portadores en Uruguay evidencia la necesidad de implementar estrategias que permitan eliminar gradualmente el defecto de la población.
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SUMMARY. Brachyspina syndrome is a hereditary recessive disease of recent identification in the Holstei... Presentar Todo |
Palabras claves : |
Artificial insemination; BOS TAURUS; Fertility; Hereditary disease. |
Thesagro : |
ENFERMEDADES HEREDITARIAS; FERTILIDAD; INSEMINACIÓN ARTIFICIAL. |
Asunto categoría : |
L73 Enfermedades de los animales |
URL : |
https://bibliotecavirtual.unl.edu.ar/publicaciones/index.php/FAVEveterinaria/article/view/9523/12826
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Marc : |
LEADER 04114naa a2200325 a 4500 001 1061752 005 2021-02-23 008 2020 bl uuuu u00u1 u #d 022 $a1666-938X (impresa); 2362-5589 (digital) 024 7 $a10.14409/favecv.v19i2.9523$2DOI 100 1 $aARTIGAS, R. 245 $aIdentificación por catálogo y detección molecular de bovinos Holstein portadores de braquiespina en Uruguay. [Identification through sire catalogues analysis and molecular detection of brachyspina carriers in Uruguayan Holstein.]$h[electronic resource] 260 $c2020 500 $aArticle history: Recibido 3 Marzo 2020; Aceptado 1 Agosto 2020; Disponible en línea 14 Agosto 2020. Editor: P. Beldomenico. Esta obra está bajo licencia internacional Creative Commons Reconocimiento-NoComercial-CompartirIgual 4.0. 520 $aRESUMEN. El síndrome de braquiespina es una condición genética de la raza Holstein, detectada en el año 2006. Es causado por una deleción de 3.3 Kb en el gen FANCI localizado en el cromosoma bovino 21. La mutación fue identificada en poblaciones de Holstein de Europa, América del Norte y Asia. Dada la importancia económica del defecto y su amplia distribución mundial, el objetivo de este trabajo ha sido la identificación de animales portadores en el núcleo de selección genética de la raza en Uruguay y el diagnóstico molecular del alelo deletéreo en animales del rodeo nacional. En el presente estudio se analizaron 2598 registros de toros Holstein del catálogo de padres del sistema de evaluación genética lechera, los registros de toros pertenecientes a los catálogos de semen Holstein disponible para Uruguay de los años 2014 al 2018; y 71 vacas pertenecientes al rodeo general. Se encontraron 28 toros portadores de braquiespina de un total de 377 toros con información genética del catálogo de padres y cuatro vacas portadoras de un total de 71 genotipificadas en nuestro laboratorio. Se demostró una disminución en el ingreso de semen de animales portadores al país entre los años 2014 y 2018. La frecuencia significativa de animales portadores en Uruguay evidencia la necesidad de implementar estrategias que permitan eliminar gradualmente el defecto de la población. .-.-.-.-.-.-.-. SUMMARY. Brachyspina syndrome is a hereditary recessive disease of recent identification in the Holstein breed. It is caused by a deletion of 3.3Kb in the FANCI gene located in the bovine chromosome 21. The mutation was identified in Holstein populations of Europe, North America and Asia. Given the economic importance of the defect and its wide distribution, the objective of this work was the identification of carrier animals in the genetic selection nucleus of the breed in Uruguay and the molecular verification of the deleterious allele in animals of the national herd. In the present study, 2598 records of Holstein bulls were analyzed from the list of parents of the dairy genetic evaluation system, records of bulls belonging to the Holstein semen catalogs available for Uruguay from 2014 to 2018; and 71 cows belonging to the general herd. Twenty-eight brachyspina carrier bulls were found of a total of 377 bulls with genetic information from the list of parents and four carrier cows of a total of 71 genotyped in our laboratory. A decrease in the income of semen from carrier animals to the country between 2014 and 2018 was demonstrated. The significant frequency of carrier animals in Uruguay evidences the need to implement strategies to gradually eliminate the population defect. 650 $aENFERMEDADES HEREDITARIAS 650 $aFERTILIDAD 650 $aINSEMINACIÓN ARTIFICIAL 653 $aArtificial insemination 653 $aBOS TAURUS 653 $aFertility 653 $aHereditary disease 700 1 $aFEDERICI, M. 700 1 $aVÁZQUEZ, N. 700 1 $aALCÁNTARA, M. 700 1 $aRAMÍREZ, M. 700 1 $aGUERRA, S. 700 1 $aDUTRA, F. 700 1 $aLLAMBÍ, S. 773 $tFAVE Sección Ciencias Veterinarias, 2020, volume 19 (2), 50-54. OPEN ACCESS. DOI: https://doi.org/10.14409/favecv.v19i2.9523
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Registro completo
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Biblioteca (s) : |
INIA Las Brujas; INIA Tacuarembó. |
Fecha actual : |
21/12/2020 |
Actualizado : |
21/12/2020 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Circulación / Nivel : |
Internacional - -- |
Autor : |
FERRINHO, A.M.; PERIPOLLI, E.; BANCHERO, G.; PEREIRA, A.S.C.; BRITO, G.; LA MANNA, A.; FERNANDEZ, E.; MONTOSSI, F.; KLUSKA, S.; MUELLER, L.F.; BERCHIELLI, T.T.; BALDI, F. |
Afiliación : |
ADRIELLE MATIAS FERRINHO, Faculdade de Zootecnia e Engenharia de Alimentos, Universidade de São Paulo, Pirassununga, SP, Brazil; Instituto Nacional de Ciência e Tecnologia-Ciencia Animal, Viçosa, MG, Brazil; ELISA PERIPOLLI, Departamento de Zootecnia, Faculdade de Ciências Agrárias e Veterinárias, Universidade Estadual Paulista, Jaboticabal, SP, Brazil; GEORGGET ELIZABETH BANCHERO HUNZIKER, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ANGÉLICA SIMONE CRAVO PEREIRA, Faculdade de Zootecnia e Engenharia de Alimentos, Universidade de São Paulo, Pirassununga, SP, Brazil; GUSTAVO WALTER BRITO DIAZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ALEJANDRO FRANCISCO LA MANNA ALONSO, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ENRIQUE GENARO FERNANDEZ RODRIGUEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; FABIO MARCELO MONTOSSI PORCHILE, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; SABRINA KLUSKA, Departamento de Zootecnia, Faculdade de Ciências Agrárias e Veterinárias, Universidade Estadual Paulista, Jaboticabal, SP, Brazil; LENISE FREITAS MUELLER, Faculdade de Zootecnia e Engenharia de Alimentos, Universidade de São Paulo, Pirassununga, SP, Brazil; TELMA TERESINHA BERCHIELLI, Dpto Zootecnia, Fac. Ciências Agrárias e Veterinárias, Univ. Estadual Paulista, SP, Brazil; Conselho Nacional de Desenvolvimento Cientifico e Tecnologico, Brasília, DF, Brazil; Instituto Nacional de Ciência e Tecnologia-Ciencia Animal, Viçosa, MG, Brazil; FERNANDO SEBASTIAN BALDI REY, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
Título : |
Effect of growth path on carcass and meat-quality traits of Hereford steers finished on pasture or in feedlot. |
Fecha de publicación : |
2020 |
Fuente / Imprenta : |
Animal Production Science, 2020, Volume 60, Issue 2, Pages 323-332. Doi: https://doi.org/10.1071/AN18075 |
ISSN : |
1836-0939 |
DOI : |
10.1071/AN18075 |
Idioma : |
Inglés |
Notas : |
Article history: Received 30 January 2018 / Accepted 14 May 2019 / Published online 4 December 2019.
Funding information:
This work was supported by Sao Paulo Research Foundation (FAPESP grant #2016/22022-4 and #2011/21241-0), Brazil, and the National Institute of Agricultural research (INIA) of Uruguay. |
Contenido : |
ABSTRACT.
The objective of this study was to evaluate the effect of nutritional management during rearing and finishing phases on beef fatty acid composition, and carcass and beef quality traits of Hereford cattle. The study used 240 castrated male calves weaned at 8 months of age, and with an average weight of 170 ± 17 kg. After weaning, the experiment was divided in to three phases in a 4 × 2 factorial design: a 93-day winter period with four treatment groups (on pasture or in feedlot and at high or low feeding levels); a 196-day compensatory-growth phase on pasture; and a finishing phase either on pasture or in feedlot. Animals were slaughtered when each group attained a mean liveweight of 500 kg. The winter growth × finishing management interaction significantly affected hot carcass weight (P = 0.0029). There was no differences observed for feedlot-finished steers, but for pasture-finished steers, those pasture-reared had higher hot carcass weight (kg) than those feedlot-reared (low pasture 256.30 ± 1.60, high pasture 253.72 ± 1.60, low feedlot 249.85 ± 1.66, high feedlot 247.60 ± 1.62). Feedlot-finished steers showed higher (P < 0.05) mean values than pasture-finished steers for ribeye area (55.61 ± 0.69 cm2 vs 53.18 cm2), backfat thickness (8.62 ± 0.32 mm vs 6.21 mm), marbling score (237.97 ± 13.06 vs 171.70) and final pH (5.53 ± 0.02 vs 5.48). Additionally, feedlot-finished steers raised in feedlot during the winter-growth period displayed the heaviest hindquarter cuts. Meat from pasture-finished steers had lower (P < 0.05) shear-force values than from feedlot-finished cattle (2.95 ± 0.18 vs 3.66 ± 0.17 kg), and when reared on either high or low pasture during winter-growth, they showed the highest (P < 0.05) conjugated linoleic acid (cis-9, trans-11) and n-3 polyunsaturated fatty acid concentrations. In conclusion, growing and finishing cattle on pasture improved the carcass yield of retail cuts because of low fat concentration, and improved the nutritional and health value of the beef fatty acid profile.
© 2020 CSIRO. MenosABSTRACT.
The objective of this study was to evaluate the effect of nutritional management during rearing and finishing phases on beef fatty acid composition, and carcass and beef quality traits of Hereford cattle. The study used 240 castrated male calves weaned at 8 months of age, and with an average weight of 170 ± 17 kg. After weaning, the experiment was divided in to three phases in a 4 × 2 factorial design: a 93-day winter period with four treatment groups (on pasture or in feedlot and at high or low feeding levels); a 196-day compensatory-growth phase on pasture; and a finishing phase either on pasture or in feedlot. Animals were slaughtered when each group attained a mean liveweight of 500 kg. The winter growth × finishing management interaction significantly affected hot carcass weight (P = 0.0029). There was no differences observed for feedlot-finished steers, but for pasture-finished steers, those pasture-reared had higher hot carcass weight (kg) than those feedlot-reared (low pasture 256.30 ± 1.60, high pasture 253.72 ± 1.60, low feedlot 249.85 ± 1.66, high feedlot 247.60 ± 1.62). Feedlot-finished steers showed higher (P < 0.05) mean values than pasture-finished steers for ribeye area (55.61 ± 0.69 cm2 vs 53.18 cm2), backfat thickness (8.62 ± 0.32 mm vs 6.21 mm), marbling score (237.97 ± 13.06 vs 171.70) and final pH (5.53 ± 0.02 vs 5.48). Additionally, feedlot-finished steers raised in feedlot during the winter-growth period displayed the heaviest hindquarter cut... Presentar Todo |
Palabras claves : |
Carry-over effects; Fatty-acid profile; Finishing system; Growth-out. |
Asunto categoría : |
L01 Ganadería |
Marc : |
LEADER 03376naa a2200337 a 4500 001 1060696 005 2020-12-21 008 2020 bl uuuu u00u1 u #d 022 $a1836-0939 024 7 $a10.1071/AN18075$2DOI 100 1 $aFERRINHO, A.M. 245 $aEffect of growth path on carcass and meat-quality traits of Hereford steers finished on pasture or in feedlot.$h[electronic resource] 260 $c2020 500 $aArticle history: Received 30 January 2018 / Accepted 14 May 2019 / Published online 4 December 2019. Funding information: This work was supported by Sao Paulo Research Foundation (FAPESP grant #2016/22022-4 and #2011/21241-0), Brazil, and the National Institute of Agricultural research (INIA) of Uruguay. 520 $aABSTRACT. The objective of this study was to evaluate the effect of nutritional management during rearing and finishing phases on beef fatty acid composition, and carcass and beef quality traits of Hereford cattle. The study used 240 castrated male calves weaned at 8 months of age, and with an average weight of 170 ± 17 kg. After weaning, the experiment was divided in to three phases in a 4 × 2 factorial design: a 93-day winter period with four treatment groups (on pasture or in feedlot and at high or low feeding levels); a 196-day compensatory-growth phase on pasture; and a finishing phase either on pasture or in feedlot. Animals were slaughtered when each group attained a mean liveweight of 500 kg. The winter growth × finishing management interaction significantly affected hot carcass weight (P = 0.0029). There was no differences observed for feedlot-finished steers, but for pasture-finished steers, those pasture-reared had higher hot carcass weight (kg) than those feedlot-reared (low pasture 256.30 ± 1.60, high pasture 253.72 ± 1.60, low feedlot 249.85 ± 1.66, high feedlot 247.60 ± 1.62). Feedlot-finished steers showed higher (P < 0.05) mean values than pasture-finished steers for ribeye area (55.61 ± 0.69 cm2 vs 53.18 cm2), backfat thickness (8.62 ± 0.32 mm vs 6.21 mm), marbling score (237.97 ± 13.06 vs 171.70) and final pH (5.53 ± 0.02 vs 5.48). Additionally, feedlot-finished steers raised in feedlot during the winter-growth period displayed the heaviest hindquarter cuts. Meat from pasture-finished steers had lower (P < 0.05) shear-force values than from feedlot-finished cattle (2.95 ± 0.18 vs 3.66 ± 0.17 kg), and when reared on either high or low pasture during winter-growth, they showed the highest (P < 0.05) conjugated linoleic acid (cis-9, trans-11) and n-3 polyunsaturated fatty acid concentrations. In conclusion, growing and finishing cattle on pasture improved the carcass yield of retail cuts because of low fat concentration, and improved the nutritional and health value of the beef fatty acid profile. © 2020 CSIRO. 653 $aCarry-over effects 653 $aFatty-acid profile 653 $aFinishing system 653 $aGrowth-out 700 1 $aPERIPOLLI, E. 700 1 $aBANCHERO, G. 700 1 $aPEREIRA, A.S.C. 700 1 $aBRITO, G. 700 1 $aLA MANNA, A. 700 1 $aFERNANDEZ, E. 700 1 $aMONTOSSI, F. 700 1 $aKLUSKA, S. 700 1 $aMUELLER, L.F. 700 1 $aBERCHIELLI, T.T. 700 1 $aBALDI, F. 773 $tAnimal Production Science, 2020, Volume 60, Issue 2, Pages 323-332. Doi: https://doi.org/10.1071/AN18075
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