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Registros recuperados : 5 | |
1. |  | PESSOA, D. A. N.; SILVA, L. C. A.; MENDONÇA, F. S.; ALMEIDA, V. M.; LOPES, J. R. G.; ALBUQUERQUE, L. G.; SILVA, A. .; RIET-CORREA, F. Evaluation of resistance to natural poisoning by Amorimia septentrionalis in goats which had received sodium monofluoroacetate degrading bacteria. [Avaliação da resistência à intoxicação natural por Amorimia septentrionalis em caprinos que receberam bactérias degradadoras de monofluoroacetato de sódio.] Pesquisa Veterinaria Brasileira 38(10):1913-1917, 1 October 2018. OPEN ACCESS Article history: Received on June 5, 2018. // Accepted for publication on June 13, 2018.Biblioteca(s): INIA La Estanzuela. |
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2. |  | ALMEIDA, T.H.S.; ALBUQUERQUE, R.; ALMEIDA, V.M.; SILVA FILHO, G.B.; CHAVES, H.A.; FREITAS, S.H.; RIET-CORREA, F.; MENDONÇA, F.S. Poisoning by Thiloa glaucocarpa (Combretaceae) in cattle in the semiarid regions of Paraíba and Pernambuco, Brazil. Brazilian Journal of Veterinary Pathology, November 2017, Volume 10, Issue 3, Pages 111-116. OPEN ACCESS Article history: Submitted July 5, 2017 / Accepted August 13. 2017.Biblioteca(s): INIA Las Brujas. |
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3. |  | MENDONÇA, F.S.; NASCIMENTO , N.C. F.; ALMEIDA, V.M.; BRAGA, T.C.; RIBEIRO, D.P.; CHAVES, H.A.S.; SILVA FILHO , G.B.; RIET-CORREA, F. An outbreak of poisoning by Kalanchoe blossfeldiana in cattle in northeastern Brazil. Tropical Animal Health and Production, 2018, v. 50, n. 3, p. 693-696. Article History: Received: 17 February 2017 /Accepted: 30 October 2017 / Published online: 16 November 2017.Biblioteca(s): INIA La Estanzuela. |
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4. |  | BRITO, L.B; RIET-CORREA, F.; ALMEIDA, V.M.; SILVA FILHO, G.B.; CHAVES, A.S.; BRAGA, T.C.; NETO, J.E.; MENDONÇA, F.S. Spontaneous poisoning by Ricinus communis leaves (Euphorbiaceae) in goats. [Intoxicação espontânea por folhas de Ricinus communis (Euphorbiaceae) em caprinos.] Pesquisa Veterinaria Brasileira, 1 February 2019, Volume 39, Issue 2, Pages 123-128. OPEN ACCESS. Doi: http://dx.doi.org/10.1590/1678-5150-pvb-5992 Article history: Received: August 05, 2018 / Accepted: October 25, 2018.
Corresponding autor: Fábio S. Mendonça - fabio.mendonca@pq.cnpq.brBiblioteca(s): INIA La Estanzuela. |
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5. |  | SOUSA, R.F.A.; ALMEIDA, V.M.; NETO, J.E.; NASCIMENTO, C.W.A.; MEDEIROS, G.X.; MEDEIROS, R.M.T.; RIET-CORREA, F.; MENDONÇA, F.S. Laryngeal neuropathy in adult goats with copper deficiency. Veterinary Pathology, 2017, v. 54, n.4., p.676-682. Article history: Article first published online: December 14, 2016.//Issue published: July 1, 2017.Biblioteca(s): INIA La Estanzuela. |
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Registros recuperados : 5 | |
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 | Acceso al texto completo restringido a Biblioteca INIA La Estanzuela. Por información adicional contacte bib_le@inia.org.uy. |
Registro completo
|
Biblioteca (s) : |
INIA La Estanzuela. |
Fecha actual : |
11/07/2017 |
Actualizado : |
30/01/2020 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Circulación / Nivel : |
Internacional - -- |
Autor : |
SOUSA, R.F.A.; ALMEIDA, V.M.; NETO, J.E.; NASCIMENTO, C.W.A.; MEDEIROS, G.X.; MEDEIROS, R.M.T.; RIET-CORREA, F.; MENDONÇA, F.S. |
Afiliación : |
R. F. A. SOUSA, Programa de Pós-Graduação em Ciência Veterinária, Universidade Federal Rural de Pernambuco, Brasil.; V.M. ALMEIDA, Programa de Pós-Graduação em Ciência Veterinária, Universidade Federal Rural de Pernambuco, Brasil; J. E. NETO, Programa de Pós-Graduação em Ciência Veterinária, Universidade Federal Rural de Pernambuco, Brasil; C W A NASCIMENTO, Departamento de Agronomia, Universidade Federal Rural de Pernambuco, Recife, Pernambuco, Brazil.; G. X. MEDEIROS, Hospital Veterinario, CSTR, Patos, Brazil.; R. M. T. MEDEIROS, Hospital Veterinario, CSTR, Patos, Brazil.; FRANKLIN RIET-CORREA AMARAL, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; F.S. MENDONÇA, Laboratorio de Diagnostico Animal, DMFA/UFRPE, Recife, Pernambuco, Brasil. |
Título : |
Laryngeal neuropathy in adult goats with copper deficiency. |
Fecha de publicación : |
2017 |
Fuente / Imprenta : |
Veterinary Pathology, 2017, v. 54, n.4., p.676-682. |
DOI : |
10.1177/0300985816681410 |
Idioma : |
Inglés |
Notas : |
Article history: Article first published online: December 14, 2016.//Issue published: July 1, 2017. |
Contenido : |
Abstract:
The aim of this study was to elucidate the cause of a neurological syndrome characterized by stridor in adult goats with clinical
signs of copper deficiency. The main clinical signs consisted of apathy, emaciation, pale mucous membranes, mucous nasal discharge,
dyspnea, severe achromotrichia, diffuse alopecia, torpor, ataxia, and stridor. When the goats were forced to move, the
stridor increased. In a herd of 194 Toggenburg goats, 10 adult goats with clinical signs of copper deficiency were removed from
the herd and divided into 2 groups: group 1, which consisted of 4 nannies and 1 buck with stridor, and group 2, which consisted of
4 nannies and 1 buck without stridor. Group 3, used as a control, consisted of 5 adult goats from another flock without any clinical
signs of disease. The mean serum copper concentrations were 1.3 + 0.3 mmol/L in group 1, 8.1 + 1.1 mmol/L in group 2, and
11.3 + 2.2 mmol/L in group 3. The mean serum iron concentrations were 42.3 + 14.2 mmol/L in group 1, 39.1 + 8.2 mmol/L in
group 2, and 20.6+ 6.1 mmol/L in group 3. The main histological lesions in goats from group 1 were axonal degeneration of the
recurrent laryngeal nerves and atrophy of the muscles of vocal folds and of the dorsal cricoarytenoid and right and left cricothyroid
muscles. Goats with ataxia had neuronal degeneration and necrosis of cerebellar Purkinje cells and of the cranial
cervical ganglion. We concluded that the stridor was caused by axonal degeneration of the recurrent laryngeal nerves due to the
severe copper deficiency. MenosAbstract:
The aim of this study was to elucidate the cause of a neurological syndrome characterized by stridor in adult goats with clinical
signs of copper deficiency. The main clinical signs consisted of apathy, emaciation, pale mucous membranes, mucous nasal discharge,
dyspnea, severe achromotrichia, diffuse alopecia, torpor, ataxia, and stridor. When the goats were forced to move, the
stridor increased. In a herd of 194 Toggenburg goats, 10 adult goats with clinical signs of copper deficiency were removed from
the herd and divided into 2 groups: group 1, which consisted of 4 nannies and 1 buck with stridor, and group 2, which consisted of
4 nannies and 1 buck without stridor. Group 3, used as a control, consisted of 5 adult goats from another flock without any clinical
signs of disease. The mean serum copper concentrations were 1.3 + 0.3 mmol/L in group 1, 8.1 + 1.1 mmol/L in group 2, and
11.3 + 2.2 mmol/L in group 3. The mean serum iron concentrations were 42.3 + 14.2 mmol/L in group 1, 39.1 + 8.2 mmol/L in
group 2, and 20.6+ 6.1 mmol/L in group 3. The main histological lesions in goats from group 1 were axonal degeneration of the
recurrent laryngeal nerves and atrophy of the muscles of vocal folds and of the dorsal cricoarytenoid and right and left cricothyroid
muscles. Goats with ataxia had neuronal degeneration and necrosis of cerebellar Purkinje cells and of the cranial
cervical ganglion. We concluded that the stridor was caused by axonal degeneration of the recurren... Presentar Todo |
Palabras claves : |
COOPER DEFICIENCY; DEFICIENCIA DE COBRE; DEFICIENCIA DE HIERRO; IRON; LARYNGEAL; MUSCLE; NERVOUS SYSTEM; PARALISIS; PARALYSIS; RUMINANTS; SISTEMA NERVIOSO; SNORING DISEASE; STRIDOR. |
Thesagro : |
OVEJAS; RUMIANTES. |
Asunto categoría : |
E16 Enfermedades de los animales |
Marc : |
LEADER 02719naa a2200409 a 4500 001 1057372 005 2020-01-30 008 2017 bl uuuu u00u1 u #d 024 7 $a10.1177/0300985816681410$2DOI 100 1 $aSOUSA, R.F.A. 245 $aLaryngeal neuropathy in adult goats with copper deficiency.$h[electronic resource] 260 $c2017 500 $aArticle history: Article first published online: December 14, 2016.//Issue published: July 1, 2017. 520 $aAbstract: The aim of this study was to elucidate the cause of a neurological syndrome characterized by stridor in adult goats with clinical signs of copper deficiency. The main clinical signs consisted of apathy, emaciation, pale mucous membranes, mucous nasal discharge, dyspnea, severe achromotrichia, diffuse alopecia, torpor, ataxia, and stridor. When the goats were forced to move, the stridor increased. In a herd of 194 Toggenburg goats, 10 adult goats with clinical signs of copper deficiency were removed from the herd and divided into 2 groups: group 1, which consisted of 4 nannies and 1 buck with stridor, and group 2, which consisted of 4 nannies and 1 buck without stridor. Group 3, used as a control, consisted of 5 adult goats from another flock without any clinical signs of disease. The mean serum copper concentrations were 1.3 + 0.3 mmol/L in group 1, 8.1 + 1.1 mmol/L in group 2, and 11.3 + 2.2 mmol/L in group 3. The mean serum iron concentrations were 42.3 + 14.2 mmol/L in group 1, 39.1 + 8.2 mmol/L in group 2, and 20.6+ 6.1 mmol/L in group 3. The main histological lesions in goats from group 1 were axonal degeneration of the recurrent laryngeal nerves and atrophy of the muscles of vocal folds and of the dorsal cricoarytenoid and right and left cricothyroid muscles. Goats with ataxia had neuronal degeneration and necrosis of cerebellar Purkinje cells and of the cranial cervical ganglion. We concluded that the stridor was caused by axonal degeneration of the recurrent laryngeal nerves due to the severe copper deficiency. 650 $aOVEJAS 650 $aRUMIANTES 653 $aCOOPER DEFICIENCY 653 $aDEFICIENCIA DE COBRE 653 $aDEFICIENCIA DE HIERRO 653 $aIRON 653 $aLARYNGEAL 653 $aMUSCLE 653 $aNERVOUS SYSTEM 653 $aPARALISIS 653 $aPARALYSIS 653 $aRUMINANTS 653 $aSISTEMA NERVIOSO 653 $aSNORING DISEASE 653 $aSTRIDOR 700 1 $aALMEIDA, V.M. 700 1 $aNETO, J.E. 700 1 $aNASCIMENTO, C.W.A. 700 1 $aMEDEIROS, G.X. 700 1 $aMEDEIROS, R.M.T. 700 1 $aRIET-CORREA, F. 700 1 $aMENDONÇA, F.S. 773 $tVeterinary Pathology, 2017$gv. 54, n.4., p.676-682.
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